Zinc ranks 34 of 461 Obesity treatments based on 15 research articles we have analyzed from 520 relevant articles we identified. The relationship is in the bottom 13% of our analyses.

About Zinc and Obesity

Obesity is a medical condition in which excess body fat has accumulated to the extent that it may have a negative effect on health, leading to reduced life expectancy and/or increased health problems. In Western countries, people are considered obese when their body mass index (BMI), a measurement obtained by dividing a person's weight by the square of the person's height, exceeds 7001300000000000000â™ 30 kg/m, with the range 25-7001300000000000000â™ 30 kg/m defined as overweight. Some East Asian countries use stricter criteria.1

Zinc, in commerce also spelter, is a chemical element with symbol Zn and atomic number 30. It is the first element of group 12 of the periodic table. In some respects zinc is chemically similar to magnesium: its ion is of similar size and its only common oxidation state is +2. Zinc is the 24th most abundant element in Earth's crust and has five stable isotopes. The most common zinc ore is sphalerite (zinc blende), a zinc sulfide mineral. The largest mineable amounts are found in Australia, Asia, and the United States. Zinc production includes froth flotation of the ore, roasting, and final extraction using electricity (electrowinning).2

There have been 33,385 reported side effects for Zinc in the US. The 5 most reported: Nausea, Dyspnoea, Anosmia, Diarrhoea, Ageusia.

The average price we found for Zinc products was $10.74. The lowest price we found was $10.74

What researchers say about Zinc as Obesity treatment

Researchers report:

  • Besides lifestyle modification, [Zinc] supplementation might be considered as a useful and safe additional intervention treatment for improvement of cardiometabolic risk factors related to childhood [Obesity].1
  • Several biomarkers are being suggested as the link between [Obesity], insulin resistance, cardiovascular disease and metabolic syndrome, such as tumor necrosis factor alfa, interleukin-6 and -18, angiotensinogen, transforming grow factor beta, plasminogen activator inhibitor-1, leptin, resistin, C-reactive protein, serum amyloid A, sialic acid, fibrinogen, markers of endothelial dysfunction (von Willebrand factor, ICAMs, VCAMs), complement factor 3, haptoglobin, [Zinc]-alpha2-glycoprotein, eotaxin, visfatin, apelin, alpha1-antitrypsin, vaspin, omentin, retinol binding protein 4, ceruloplasmin, adiponectin and desnutrin.2
  • Serum iron (sFe), and ferritin (sFert), transferrin saturation index (TSI), plasma [Zinc] and copper (pZn, pCu), and erythrocyte [Zinc] content (eZn) were measured in 55 [Obesity] children and adolescents (28 males and 27 females) before and after a 13-wk treatment with a hypocaloric balanced diet (HCBD, 22 subjects) or a 10-wk treatment with a protein sparing modified fast diet (PSMF, 33 subjects).3
  • We therefore examined the gene-expression levels of a number of [Zinc]-transporting proteins in adipose tissue, comparing subcutaneous fat with visceral fat from lean and [Obesity] humans.4
  • In this investigation, we have examined the role of 9 most significant SNPs reported in GWA studies: [peroxisome proliferator-activated receptor gamma 2 (PPARG2; rs 1801282); insulin-like growth factor two binding protein 2 (IGF2BP2; rs 4402960); cyclin-dependent kinase 5, a regulatory subunit-associated protein1-like 1 (CDK5; rs7754840); a [Zinc] transporter and member of solute carrier family 30 (SLC30A8; rs13266634); a variant found near cyclin-dependent kinase inhibitor 2A (CDKN2A; rs10811661); hematopoietically expressed homeobox (HHEX; rs 1111875); transcription factor-7-like 2 (TCF7L2; rs 10885409); potassium inwardly rectifying channel subfamily J member 11(KCNJ11; rs 5219); and fat mass [Obesity]-associated gene (FTO; rs 9939609)].5

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Research last analyzed March 03, 2015 @8:53AM.